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Adplatt (Clopidogrel)
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Adplatt

Adplatt is an antiplatelet medicine, or blood thinner. It works by preventing cells called platelets from sticking together and helps to prevent your blood from clotting inside veins and arteries. This helps the blood flow freely around your body thereby preventing a heart attack or stroke (or deep vein thrombosis or pulmonary embolism).

Other names for this medication:
Aclotil, Antiban, Asogrel, Blothin, Carpigrel, Cenoza, Clavix, Clopact, Clopicure, Clopione, Clopirad, Clopisa, Clopitab, Cloplat, Clopod, Clospir, Closprez, Clotnil, Clotsafe, Combiplet, Compivas, Dospin, Ecosprin, Grelet, Kabitrol, Livigrel, Neogrel, Oprin, Orawis, Platloc, Plodrel, Psygrel, Rozanex, Starclop, Thrombosprin, Torpido, Torplatt, Welgrel, Zeter, Zogrell

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Also known as: Clopidogrel

Description

Adplatt (clopidogrel) prevents platelets in your blood from sticking together to form an unwanted blood clot that could block an artery.

Adplatt is used to lower your risk of having a stroke, blood clot, or serious heart problem after you've had a heart attack, severe chest pain (angina), or circulation problems.

Dosage

Usual Adult Dose for Acute Coronary Syndrome:

Unstable Angina (UA)/Non-ST-Elevation Myocardial Infarction (NSTEMI): -Loading dose: 300 mg orally once -Maintenance dose: 75 mg orally once a day -Duration of therapy: Optimal duration unknown. ST-Elevation Myocardial Infarction (STEMI): -Loading dose: 300 mg orally once (OPTIONAL) -Maintenance dose: 75 mg orally once a day, with or without thrombolytics -Duration of therapy: Optimal duration unknown. Comments: -Administer this drug in combination with aspirin 75 mg to 325 mg orally once a day. -The benefit of this drug for patients who undergo primary percutaneous coronary intervention is unknown. Uses: Prevention of atherothrombotic events in patients with non-ST-segment elevation acute coronary syndrome (UA, NSTEMI, those who are to be managed medically, those who are to be managed with coronary revascularization) and STEMI.

Usual Adult Dose for Ischemic Stroke:

75 mg orally once a day Uses: Prevention of atherothrombotic events in patients with a history of recent myocardial infarction, recent stroke, or established peripheral arterial disease.

Usual Adult Dose for Myocardial Infarction:

75 mg orally once a day Uses: Prevention of atherothrombotic events in patients with a history of recent myocardial infarction, recent stroke, or established peripheral arterial disease.

Usual Adult Dose for Peripheral Arterial Disease:

75 mg orally once a day Uses: Prevention of atherothrombotic events in patients with a history of recent myocardial infarction, recent stroke, or established peripheral arterial disease.

Detailed Adplatt dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222. Overdose can cause excessive bleeding.

Storage

Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children in a container that small children cannot open.

Side effects

The most common side effects associated with Adplatt are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

You should not use Adplatt if you are allergic to clopidogrel, or if you have:

any active bleeding; or a stomach ulcer or bleeding in the brain (such as from a head injury).

Tell your doctor if you have ever had:

an ulcer in your stomach or intestines; or a bleeding disorder or blood clotting disorder.

Adplatt may not work as well if you have certain genetic factors that affect the breakdown of this medicine in your body. Your doctor may perform a blood test to make sure Adplatt is right for you.

This medicine is not expected to harm an unborn baby. Tell your doctor if you are pregnant.

You should not breastfeed while using this medicine.

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Wavelength dependence of the optically recorded action potentials in guinea pig atrial muscles. These in vivo observations provide further evidence for a direct mechanism of action by TGF-beta 1 on collagen synthesis by wound fibroblasts during soft tissue repair. The following review summarizes recent genetic findings in the congenital myopathies and outlines implications for our understanding of their pathophysiological basis and their relation to other neuromuscular disorders. Dynamics of multiple nuclei in Ashbya gossypii hyphae depend on the control of cytoplasmic microtubules length by Bik1, Kip2, Kip3, and not on a capture/shrinkage mechanism. More research is needed on nutritional intake of antioxidative nutrients to determine whether they can be used to reduce oxidative stress. These families were examined clinically and electrophysiologically. The results show that the cerebellar nuclei project to the deep layers of the superior colliculus, especially contralaterally. A death occurs when the accumulated wear exceeds the initial random resource. The Gordon Wilson Lecture: atherosclerosis--a response to injury gone awry.

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