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Atwin (Atenolol)
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Atwin

Atwin is a beta blocker that works specifically on the heart. It works by slowing down the heart rate and makes the heart more efficient at pumping blood around the body. Atwin is also used to relieve the symptoms caused by an overactive thyroid gland.

Other names for this medication:
Adbeta, Amdonex, Amlip, Amloact, Amlobet, Amloman, Amloray, Amlozed, Amnorm, Amodin, Amtas, Andinx, Atedine, Atepose, Atzee, Bardateno, Betanol, Biocard, Dibeta, Dipress, Licopin, Loten, Odipin, Olpine, Opime, Samstar, Systonorm, Telol, Tenocab, Tenochek, Ziblok

Similar Products:
Theolair, Exelon, Strattera, Differin, Lexapro, Lopressor, Asacol, Klonopin, Detrol, Benemid, Neurophas, Emispan, Amloheart, Cordinate, Nitrofix,

Also known as: Atenolol

Description

Atenolol injection is used to reduce the risk of death from an acute heart attack. It is given to people who have already had a heart attack .

This medicine is a beta-blocker. It works by affecting the response to nerve impulses in certain parts of the body, like the heart. As a result, the heart beats slower and decreases the blood pressure. When the blood pressure is lowered, the amount of blood and oxygen is increased to the heart .

This medicine is available only with your doctor's prescription .

Dosage

Usual Adult Dose of Atwin for Hypertension:

Initial dose: 50 mg orally once a day Maintenance dose: 50 to 100 mg orally once a day Maximum dose: 100 mg per day Comments: -If desired response not achieved after 1 to 2 weeks, increase to 100 mg may be beneficial. -Doses greater than 100 mg once a day did not result in significant additional antihypertensive effects. Use: For the treatment of hypertension alone or in combination with other antihypertensive agents.

Usual Adult Dose of Atwin for Angina Pectoris Prophylaxis:

Initial dose: 50 mg orally once a day -Increase to 100 mg orally once a day after 1 week if optimal response not achieved Maintenance dose: 50 to 200 mg orally once a day Maximum dose: 200 mg per day Comments: -Some patients may require 200 mg per day to attain optimal effect. Use: For the long-term management of angina pectoris due to coronary atherosclerosis.

Usual Adult Dose for Angina Pectoris:

Initial dose: 50 mg orally once a day -Increase to 100 mg orally once a day after 1 week if optimal response not achieved Maintenance dose: 50 to 200 mg orally once a day Maximum dose: 200 mg per day Comments: -Some patients may require 200 mg per day to attain optimal effect. Use: For the long-term management of angina pectoris due to coronary atherosclerosis.

Usual Adult Dose of Atwin for Myocardial Infarction:

50 mg orally twice a day or 100 mg orally once a day Comments: -If IV beta blockers are contraindicated or inappropriate, oral therapy should continue for at least 7 days post-myocardial infarction (MI). -Treatment with beta blockers post MI should generally continue for 1 to 3 years if there are no contraindications. Use: For the management of hemodynamically stable patients with definite or suspected acute myocardial infarction to reduce cardiovascular mortality.

Usual Geriatric Dose for Hypertension:

Initial dose: Consider reducing the starting dose to 25 mg orally once a day

Detailed Atwin dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include extreme weakness or lack of energy, very slow heart rate, shortness of breath, or fainting.

Storage

Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Atwin are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Appropriate studies have not been performed on the relationship of age to the effects of atenolol injection in the pediatric population. Safety and efficacy have not been established .

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Characteristic localization of non-NMDA type glutamate receptor subunits in the rat pituitary gland. Dihydrocytochalasin B did not inhibit tyrosine phosphorylation, but in its presence phosphorylation was rapidly reversed. Glomerular filtration barrier in aminoglycoside-induced nephrotoxic acute renal failure. Usefulness of plasma haloperidol levels for monitoring clinical efficacy and side effects in Alzheimer patients with psychosis and behavioral dyscontrol. The authors have encountered six cases of common iliac artery occlusion and two cases of acute vasospasm as a complication of ALIF using two different approaches to spine: hypogastric-midline-transperitoneal approach at one center and minimally invasive muscle-sparing retroperitoneal approach at the other. The elongation factor 1 A-2 isoform from rabbit: cloning of the cDNA and characterization of the protein. But2-cAMP replacement did not revert the inhibition of prolactin secretion.

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Low-dose cytarabine-induced hepatic and renal dysfunction in a patient with myelodysplastic syndrome. Especially the structural motifs deserve attention because of the structural connection to the well-known earth metal alkoxides. Mutant copies of DRR1 and DRR2 were cloned from genomic YCp50 libraries by their ability to confer drug resistance in wild-type cells. There are albendazole tablets ip 400 mg price many different sedation protocols in the literature for imaging in paediatric patients, with varying levels of success and adverse event rates. Following slaughter the bacteria were studied at the site of separating the skin, in the abdominal muscles and in the croup. The original article to which this Erratum refers was published in Journal of the Science of Food and Agriculture 87 (8): 2007, 1539-1549. Monoclonal antibodies are the dominant agents used in inhibition of biological target molecules for disease therapeutics, but there are concerns of immunogenicity, production, cost and stability. The use of intermittent pneumatic compression in the rehabilitation of stroke patients may be of clinical importance for the restoration of sensory function. The parabrachial nucleus (PB) is a major relay of noxious and non-noxious visceral sensory information from the nucleus of the solitary tract, spinal cord, and spinal trigeminal nucleus to the forebrain. aggressive therapy) as well as the differences in opinion as to the appropriate treatment between blunt and penetrating trauma are also reviewed.

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